Omerta sur la viande, un témoin parle

Les révélations de Pierre Hinard

omerta book cover image
Pierre Hinard présente des documents inouïs, et révèle tout le circuit de l’empoisonnement alimentaire ordinaire…

Pierre Hinard est un cas unique. Eleveur et fils d’éleveurs à une époque où les traditions d’élevage, d’abattage et de consommation n’étaient pas encore devenues industrielles. Près de Châteaubriant, il choie et engraisse aujourd’hui une trentaine de vaches, des Salers élevées à l’herbe et soignées aux huiles essentielles. Un puriste. Ancien créateur de marchés bio à Paris, bien avant la mode, ingénieur agronome, il a longtemps voulu croire que la qualité et l’excellence pouvaient trouver leur chemin jusqu’aux rayons viande de la grande distribution.

Un jour, il est arrivé dans une importante société d’abattage et de découpe de Loire-Atlantique qui travaillait pour Auchan, Flunch, Mc Do, William Saurin, Lustucru tout le bottin de l’agro-alimentaire ou presque. Un parcours exceptionnel et très spécialisé, des vertes prairies normandes aux steaks hachés en barquettes, via le monde secret des abattoirs. Chez Castel Viandes, Pierre Hinard découvre les dessous pas très propres des “usines à viandes” : des asticots dans la viande hachée, des pièces congelées, décongelées, recongelées, des analyses faussées, des dates truquées, du sang déversé dans les champs.

Face aux manquements répétés, des services vétérinaires absents ou corrompus et des pouvoirs publics pour le moins distraits… Et en bout de chaîne, des consommateurs lésés, méprisés… et trop souvent malades. Quand il lance l’alerte, Pierre Hinard est licencié sur le champ et cinq ans plus tard rien n’a vraiment changé. Il n’est jamais bon d’avoir raison seul contre tous. Il décide aujourd’hui de raconter de l’intérieur les dérives d’un système qui méprise le paysan et le consommateur pour ne servir que les intérêts d’une minorité sans honneur ni conscience. Il présente des documents inouïs, et révèle tout le circuit de l’empoisonnement ordinaire. Quelles conséquences pour nous, consommateurs ? Quelles sont les alternatives ? Pierre Hinard propose des solutions concrètes pour trouver et consommer des produits de qualité. Que manger redevienne un plaisir.

  • Plongée dans une industrie de la viande faisandée, LeMonde, 07.12.2014.
  • “Omerta sur la viande” : ce qui atterrit vraiment dans nos assiettes, Marianne, 26 Novembre 2014.
  • Commentaires en ligne, amazon.
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Your child will be exposed to thousands of chemicals throughout their lifetime, so why give them a head start?

Do babies need cosmetics? asks Lorraine Dallmeier

baby
After all, your child is going to be exposed to thousands of chemicals throughout their lifetime, so why give them a head start? asks Lorraine Dallmeier

” The infant cosmetics industry seems to be growing all the time, churning out new products that are “essential” for your children. Or are they? As anyone who has held a baby or toddler will know, babies and infants generally have beautiful unblemished skin. Babies’ skin is less oily than adults and their sweat glands are not yet fully developed. There is a reason that people use the expression “smooth as a baby’s bottom”! Although infant eczema is a common occurrence – it occurs in 10-15% of babies – it is often treatable (depending on various factors) and may sometimes even be caused by those skincare products that we are told our children should use, such as soaps, bath foams and body lotions…”

Continue reading Do babies need cosmetics?, herbhedgerow, 2013.

Related articles
  • Throw out the bath water?, theguardian. 8 May 2004.
  • Clarification on Opinion SCCS/1348/10 in light of the Danish clause of safeguard banning the use of parabens in cosmetic products intended for children under 3 years of age, ec.europa, 2011.

Un enfant si je peux quand je veux, vidéo documentaire de Marie Halopeau

A travers le parcours du docteur Gallo et de quelques-unes de ses patientes françaises et espagnoles, le point est fait sur la fertilité des femmes.

L’infertilité, enjeu majeur des sociétés occidentales, touche aujourd’hui un couple sur cinq. Lorsqu’elle est féminine, l’infertilité est souvent le résultat d’une maladie. Mais le rythme de vie actuel pousse également les femmes à faire un enfant de plus en plus tard. L’âge est devenu un autre facteur d’infertilité. A 40 ans, les chances de tomber enceinte naturellement sont seulement de 5 %. Si la préservation de la fertilité des femmes malades est un enjeu évident, celle des femmes saines qui souhaitent volontairement retarder le moment de leur grossesse est un nouveau défi médical et social.

Plus d’informations
  • Les Françaises font des bébés de plus en plus tard alors que leur fertilité chute à partir de 35 ans. Elles ont donc de plus en plus recours à la procréation médicalement assistée. En France, l’autoconservation de sperme est légale pour les hommes. Les femmes, elles, ne peuvent ” conserver ” leurs ovules que ” lorsque la fertilité est menacée par un traitement stérilisant ” (notamment en cas de traitement contre le cancer). Pourtant , féconder in vitro puis réimplanter leurs propres ovocytes plus jeunes, donc de meilleure qualité, permettrait d’alléger les traitements contre l’infertilité. Ce principe minimiserait aussi le recours au don d’ovocytes et éviterait à de nombreuses femmes de partir à l’étranger pour profiter de ce don et de la technique de vitrification ovocytaire. Autorisée en France depuis 2012 seulement, grâce à la révision de la loi de bioéthique, la vitrification était réclamée de longue date par les professionnels du secteur. Cette congélation ultrarapide est beaucoup plus efficace que la méthode de congélation lente utilisée auparavant. La France a en la matière des années de retard sur certains de ses voisins européens. Et notamment l’Espagne qui est un pays pionniers de la préservation de la fertilité tant pour les femmes malades pour celles qui veulent faire le choix du moment de leur maternité. Ce documentaire se propose de faire un point sur la présentation de la fertilité des femmes aujourd’hui, qu’elles soient malades ou saines, à travers le parcours du Dr Gallo et des cas de ses patientes françaises et espagnoles.
  • Video publiée le 29 juin 2014 par la chaine Tellement vrai.
  • Lire les commentaires laissés sur bamp.
Le Distilbène DES, en savoir plus

Join my PAP Smear campaign

Kathy Griffin had a public PAP Smear in 2010…

KATHY-GRIFFIN-PAP-SMEAR
Kathy Griffin had a public PAP Smear in 2010…
Watch @DES_Journal diaporama and health posters album on Flickr.

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Does Cancer start in the Womb? Predisposition to Breast Cancer due to in utero Exposure to DES, EDCs

Perturbations in the fetal environment predispose an individual to disease that only becomes apparent in adulthood

BPA-and-DES molecular images
Fetal and neonatal exposures to EDCs cause persistent alterations in the mammary glands of rodents, including pre- and neoplastic lesions, long after the exposure ended.

Abstract

We are now witnessing a resurgence of theories of development and carcinogenesis in which the environment is again being accepted as a major player in phenotype determination. Perturbations in the fetal environment predispose an individual to disease that only becomes apparent in adulthood. For example, gestational exposure to Diethylstilbestrol (DES)  resulted in clear cell carcinoma of the vagina and breast cancer.

In this review the effects of the endocrine disruptor bisphenol A (BPA) on mammary development and tumorigenesis in rodents is used as a paradigmatic example of how altered prenatal mammary development may lead to breast cancer in humans who are also widely exposed to it through plastic goods, food and drink packaging, and thermal paper receipts. Changes in the stroma and its extracellular matrix led to altered ductal morphogenesis. Additionally, gestational and lactational exposure to BPA increased the sensitivity of rats and mice to mammotropic hormones during puberty and beyond, thus suggesting a plausible explanation for the increased incidence of breast cancer..

Excerpts

“… Breast cancer risk at 40 years of age and older is 2.5 fold higher in DES-exposed women than in unexposed women of the same age. In rats, prenatal exposure to DES also resulted in increased mammary cancer incidence during adulthood when these animals were challenged with the chemical carcinogen dimethylbenzanthracene (DMBA) at puberty. DES was administered to rats at pharmacological doses to mimic its medical use… ”

“… The causal link between fetal exposure to estrogens and the development of breast cancer that was first suggested by epidemiologists has now been confirmed by the increased risk to develop breast cancer during adulthood of women exposed to DES during their fetal life. Fetal and neonatal exposures to EDCs cause persistent alterations in the mammary glands of rodents, including pre- and neoplastic lesions, long after the exposure ended. In the case of BPA, mammary neoplasias may have their origin in the altered mammary morphogenesis that occurs during fetal and neonatal exposure. The data obtained from laboratory animals support the extrapolation that exposure to BPA and other xenoestrogens during organogenesis in humans contributes to the increase in the incidence of breast cancer observed over recent decades….”

Sources and full study
  • Does cancer start in the womb? altered mammary gland development and predisposition to breast cancer due to in utero exposure to endocrine disruptors, J Mammary Gland Biol Neoplasia. PMID: 23702822, 2013 Jun;18(2):199-208. doi: 10.1007/s10911-013-9293-5. Epub 2013 May 24.
  • Full study 18(2), 199–208. doi:10.1007/s10911-013-9293-5 PMC3933259, 2013 Jun.
More DES DiEthylStilbestrol Resources

Molecular breast imaging added to screening mammography in cancer detection for dense breasts

New Breast Exam Nearly Quadruples Detection of Invasive Breast Cancers in Women with Dense Breast Tissue

MayoClinic banner image
A new breast imaging technique pioneered at Mayo Clinic nearly quadruples detection rates of invasive breast cancers in women with dense breast tissue, according to the results of a major study published this week in the American Journal of Roentgenology.

Study Abstract

OBJECTIVE
The purpose of this study was to assess the diagnostic performance of supplemental screening molecular breast imaging (MBI) in women with mammographically dense breasts after system modifications to permit radiation dose reduction.

SUBJECTS AND METHODS
A total of 1651 asymptomatic women with mammographically dense breasts on prior mammography underwent screening mammography and adjunct MBI performed with 300-MBq 99mTc-sestamibi and a direct-conversion (cadmium zinc telluride) gamma camera, both interpreted independently. The cancer detection rate, sensitivity, specificity, and positive predictive value of biopsies performed (PPV3) were determined.

RESULTS
In 1585 participants with a complete reference standard, 21 were diagnosed with cancer: two detected by mammography only, 14 by MBI only, three by both modalities, and two by neither. Of 14 participants with cancers detected only by MBI, 11 had invasive disease (median size, 0.9 cm; range, 0.5–4.1 cm). Nine of 11 (82%) were node negative, and two had bilateral cancers. With the addition of MBI to mammography, the overall cancer detection rate (per 1000 screened) increased from 3.2 to 12.0 (p < 0.001) (supplemental yield 8.8). The invasive cancer detection rate increased from 1.9 to 8.8 (p < 0.001) (supplemental yield 6.9), a relative increase of 363%, while the change in DCIS detection was not statistically significant (from 1.3 to 3.2, p =0.250). For mammography alone, sensitivity was 24%; specificity, 89%; and PPV3, 25%. For the combination, sensitivity was 91% (p < 0.001); specificity, 83% (p < 0.001); and PPV3, 28% (p = 0.70). The recall rate increased from 11.0% with mammography alone to 17.6% (p < 0.001) for the combination; the biopsy rate increased from 1.3% for mammography alone to 4.2% (p < 0.001).

CONCLUSION
When added to screening mammography, MBI performed using a radiopharmaceutical activity acceptable for screening (effective dose 2.4 mSv) yielded a supplemental cancer detection rate of 8.8 per 1000 women with mammographically dense breasts.

Sources and more information

  • Molecular Breast Imaging at Reduced Radiation Dose for Supplemental Screening in Mammographically Dense Breasts, American Journal of Roentgenology, AJR.14.13357, February 2015, Volume 204, Number 2.
  • New Breast Exam Nearly Quadruples Detection of Invasive Breast Cancers in Women with Dense Breast Tissue, mayoclinic, January 23, 2015.

March for the NHS: the @999CallforNHS #Fight4NHS and #SaveOurNHS campaigns

Watch @DES_Journal diaporama and health posters album on Flickr.

March-for-The-NHS poster
Watch @DES_Journal diaporama and health posters album on Flickr.

This winter 2015 there is a battle between those who care for people and those who only care about profits. We have to save the NHS from the greed & corruption of private companies who wish to sacrifice one of the greatest institutions of our country, recognised globally as the most cost-effective and efficient health service in the world. ” Sources: 999callfornhs.

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Low, brief BPA exposure linked to stem cells changes, lower sperm production

BPA and other estrogenic compounds hamper development of the stem cells responsible for producing sperm

plastics-and-cans image
The WSU study is the first to suggest that low, brief exposures to bisphenol-A early in life can alter the stem cells responsible for producing sperm later in life. Plastic and can bottles shame image by Michael Coghlan.

BPA and other estrogenic compounds hamper development of the stem cells responsible for producing sperm in mice, which suggests such exposure could contribute to declining sperm counts in men, according to a new study.

The study is the first to suggest that low, brief exposures to bisphenol A, or other estrogens such as those used in birth control but found as water contaminants, early in life can alter the stem cells responsible for producing sperm later in life.

Exposure to estrogensis not simply affecting sperm being produced now, but impacting the stem cell population, and that will affect sperm produced throughout the lifetime,” said Patricia Hunt, a geneticist at Washington State University who led the study.

BPA is a ubiquitous chemical found in most people and used to make polycarbonate plastic and found in some food cans and paper receipts. People also are exposed to synthetic estrogens used in birth control as they are commonly found contaminating water, even after treatment.

The U.S. Food and Drug Administration banned BPA from baby bottles in 2012 but maintains that BPA currently used in food containers and packaging is safe. And this week the European Food Safety Authority announced in a new assessment there is “no consumer health risk from bisphenol-A exposure.”

However, Hunt’s study adds to evidence that low doses of the compound may harm us.

Hunt and colleagues exposed some newborn mice to BPA and some newborn mice to a synthetic estrogen used in birth control pills and hormone therapy.

These exposures – comparable to human exposures to the compounds — caused “permanent alterations” to the stem cells responsible for sperm production, the authors wrote.

The researchers also transplanted the stem cells into unexposed mice and verified the impacts to sperm development.

It is “sobering evidence” for possible harmful impacts from short-term exposure, said Mary Ann Handel, a senior research scientist with The Jackson Laboratory, which specializes in genetics research.

Scientists previously found BPA exposure impacts mice testis size and sperm development and prostate growth. But what Hunt and colleagues did was different – they found a possible reason why these things happen: changes to the stem cells, which are vital for male reproduction.

The negative effects of estrogenic chemicals on the developing male include an expanding list of subtle changes to the developing brain, reproductive tract, and testis,” the authors wrote. “Changes in all three have the potential to induce major reproductive repercussions and … the biological underpinnings remain unclear.”

Over the past few decades, researchers have noted declining sperm counts and quality in places such as Europe, Japan and the United States. In Denmark, more than 40 percent of young men have sperm counts associated with infertility or decreased fertility.

When you show you’re impacting a stem cell – that’s a huge deal,” said University of Missouri scientist Frederick vom Saal, who was not part of the study. “This exposure could very well be the basis for transgenerational loss of sperm production.”

Sperm production is a continuous process: Once males hit puberty and start producing sperm, stem cells slowly divide and give rise to new cells to produce sperm.

And, while there are some limits in using mice and extrapolating findings to humans, the reproductive systems’ “fundamentals are the same,” Hunt said.

However, Steven Hentges of the American Chemistry Council, which represents chemical manufacturers, said in an emailed response that multiple large studies “consistently find no reproductive effects in males or females at any dose remotely close to the levels of BPA to which people are actually exposed.”

He said Hunt’s study is of “limited relevance to human health” and that the doses used were much higher than actual human exposure.

Hunt said that is not true.

The levels we used are based on previous studies and produce very low levels in blood that are lower than those reported in humans,” Hunt said.

Vom Saal said it’s important in future studies to see if the stem cell changes from exposure are passed to future generations. Evidence suggests that estrogenic compounds appear to alter the ability of genes to function properly, a phenomenon referred to as epigenetic changes.

When such changes happen, it can mean similar problems in sperm production for future generations. And “since most people are consistently exposed to BPA and other estrogenic compounds, each generation could have it a bit worse,” vom Saal said.

Hunt and colleagues did run into one problem – there are secondary impacts, such as fluid retention, which make it difficult to take the stem cell research to the next level and look at correlations in sperm cell counts and measures of reproductive ability.

Exposure is not just affecting cells in testis but the whole animal,” Hunt said.

Hunt admits this is “complicated genetics stuff,” but said the consequences are quite important.

This implicates cells way upstream” and could mean problems for “subsequent generations after exposure,” she said.

This article, written by Brian Bienkowski, originally ran at Environmental Health News, a news source published by Environmental Health Sciences, a nonprofit media company.

Pollution et perturbateurs endocriniens: quels risques?

Quels sont leurs effets des perturbateurs endocriniens sur la santé ? Quelles sont les méthodes de prévention ? Où en est la recherche dans ce domaine ?

Diverses études scientifiques ont établi un lien entre certaines pathologies et la présence de polluants dans le sang ou les urines. Une partie de ces polluants, les perturbateurs endocriniens, agissent sur le système hormonal et altèrent le fonctionnement de l’organisme d’un individu et de ses descendants.

Quels sont leurs effets sur la santé ?
Quelles sont les méthodes de prévention ?
Où en est la recherche dans ce domaine ?

Quelques corrections sur le contenu – de 19:19 à 23:05
  • La molécule de synthèse DES veut dire Di Ethyl Stylbestrol (et non pas distilbène).
    La molécule DES a été commercialisée par divers laboratoires de différents pays sous plus de 200 noms différents. “Distilbène” est juste le nom d’un médicament sous lequel le DES a été produit et vendu en France.
  • En 1971 la FDA s’est limité à contre-indiquer l’utilisation du DES chez les femmes enceintes. La FDA n’a jamais – et donc toujours pas – formellement interdit la substance…
  • Les multiples effets secondaires transgénérationnels du DES sont plus que “probables” et ne se limitent pas aux petits garçons… A ce jour les effets transmissibles du DES ont été prouvés jusqu’à la 3 ème génération, c’est à dire aux enfants et aux petits-enfants des femmes ayant consommé du DES pendant leur grossesse. Voir les résultats de l’étude 2013-2014 Réseau DES France
  • Sur ce, c’est l’intention qui compte, et je remercie vivement Rémy Slama d’avoir parlé du DES dans son exposé de conférence-débat. J’espère que le grand public et les médecins n’associent pas le Distilbène à de l’histoire passée… Tous comme ses “cousins moléculaires” – BPA et autres perturbateurs endocriniens – le DES a toujours des effets dévastateurs sur une vaste partie des enfants et progéniture à venir d’aujourd’hui.
  • Video publiée le 3 Déc 2014 par la chaine Inserm vidéos.
Le Distilbène DES, en savoir plus

PAP Smear gone wrong

Cartoon by Megan Anderson on Explosm

PAP-Smear-gone-wrong cartoon
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